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Please forward this error screen to 216. A password will be e-mailed to you. Pharmacology, as we all know, is a mind-numbing, volatile and easy-to-forget subject. Based on my personal experience, one of the most difficult things which I dealt was memorizing drug classifications, adverse effects, and contraindications.
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Anatomy and physiology are two very important subjects for students who are doing majors in biology or biomedical sciences. Netter Interactive Atlas Of Human Anatomy v3. Edition after edition, Williams Hematology has guided generations of clinicians, biomedical researchers, and trainees in many disciplines through the origins, pathophysiological mechanisms, and management of benign and malignant disorders of blood cells and coagulation proteins. Structure and Composition of the Erythrocyte Absorb former 30 Composition of Erythrocyte. Biochemistry and function of monocytes and macrophages ABSORBED into Chap 67.
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This article needs additional citations for verification. Alveolar cells, or pneumocytes, are cells lining the alveoli of the lungs. Two types of alveolar cell exist: type I alveolar cells and type II alveolar cells. Type I cells are involved in the process of gas exchange between the alveoli and blood. Organelles of type I alveolar cells such as the endoplasmic reticulum, Golgi apparatus and mitochondria are clustered around the nucleus. Type I pneumocytes are unable to replicate and are susceptible to toxic insults.
In the event of damage, type II cells can proliferate and differentiate into type I cells to compensate. Type II alveolar cells cover a small fraction of the alveolar surface area. Their function is of major importance in the secretion of pulmonary surfactant, which decreases the surface tension within the alveoli. They are also capable of cellular division, giving rise to more type II alveolar cells when the lung tissue is damaged. These cells are granular and roughly cuboidal. Type II cells start to develop at about 26 weeks of gestation, secreting small amounts of surfactant. However, adequate amounts of surfactant are not secreted until about 35 weeks of gestation – this is the main reason for increased rates of infant respiratory distress syndrome, which drastically reduces at ages above 35 weeks gestation.
Type II pneumocytes can replicate in the alveoli and will replicate to replace damaged type I cells. MUC1, a human gene associated with type II pneumocytes, has been identified as a marker in lung cancer. Alveolar epithelial type II cell: defender of the alveolus revisited. MUC1 is a novel marker for the type II pneumocyte lineage during lung carcinogenesis”. This page was last edited on 17 March 2018, at 16:38.